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TESTOSTERONE SUPPORT

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Advanced Molecular LabsTESTOSTERONE SUPPORT

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AML TEST: BEST NATURAL FREE TESTOSTERONE SUPPORT SUPPLEMENT!* HELPS SUPPORT FREE TESTOSTERONE NATURALLY* HELPS COMBAT CORTISOL* Based on Latest Scientific Research* TWO CAPSULES OF AML TEST PROVIDES: 1 DHEA (dehydroepiandrosterone) 50 mg 2 BORON 10 mg 3 FENUGREEK SEED EXTRACT (Trigonella Foenum-Graecum) 600 mg 4 TONGKAT ALI (Eurycoma Longifolia) 200 mg 5 RED WINE EXTRACT (Standardized for 30% Polyphenols.)* 6 VITAMIN D 3332/IUs 7 ZINC 30 mg Directions: For best results, take two capsules of AML TEST with an evening meal, or one hour before morning high-intensity training (HIIT) and resistance training program. * WHAT ALSO WORKS AND WHAT DOESN’T (As part of a Diet and Resistance Exercise Training Program.) D-Aspartic acid, Tribulus terrestris, stinging nettle, saw palmetto, maca root extract, damiana leaf extract, indole 3-carbinol and 3,3-diindolylmethane (DIM) are not present in AML TEST because of lack of published, scientific human research supporting increased free or total testosterone in humans.* Also, purified supplements of indole-3-carbinol and DIM are shown to be phytoestrogens, anti-androgens (blocking androgen receptors) and endocrine disruptors. They shouldn’t be taken as dietary supplements if you want to support testosterone function! If you want to get your health benefits from these compounds, then play it safe and eat your cruciferous vegetables such as broccoli, cauliflower, Brussels sprouts or asparagus. For example, the latest research has shown that D-aspartic acid decreases testosterone! Australian scientists from the University of Western Sydney, led by Geoffrey Melville, found that daily supplementation of either 3 or 6 grams of D-aspartic acid decreased total and free testosterone in resistance-trained men and had no effect on related hormones such as estrogen or sex-hormone binding globulin (Nutrition Research, 2013 & Journal of the International Society of Sports Nutrition, 2015.) Another example is Tribulus terrestris, often recommended for increasing testosterone. Tribulus terrestris is an herb that was first cultivated on the Steppes of Georgia (the country, not the state). East Bloc athletes used it to support testosterone production and improve performance. Russian scientists theorized that the herb acts like the pituitary hormone (LH) to stimulate testosterone release in the testes. The supplement has been extremely popular with bodybuilders and powerlifters for increasing performance and non-athletic men for improving sexual performance, even though little research supports its effectiveness. Well-controlled studies have found that tribulus has no effect on strength, body composition, or testosterone. (Journal of Human Kinetics, 2014). According to Halis Akturk and Todd Nippoldt from the Mayo Clinic (JAMA, 2016), “The symptoms linked to low testosterone levels can often be improved through exercise, fat loss, improved diet, reduced stress and better sleep habits.”* When it comes to diet, low-fat diets lower testosterone levels. Saturated and monounsaturated fatty acids have been shown to directly and systematically support testosterone while polyunsaturated fats, by and large, inhibit testosterone production. Arachidonic acid, an omega-6 derived polyunsaturated fatty acid, does support testosterone production, and is not recommended as a dietary supplement because it is a substrate for the inflammatory cascade and inflammation in the body. You get all the arachidonic acid you need from the diet. No need to take supplemental arachidonic acid, as it may increase inflammation and the risk of heart disease and other degenerative diseases*. The best and healthiest fats for supporting testosterone are monounsaturated fats, which are found preferably in extra-virgin olive oil, avocados and nuts. Not saturated fats, which can increase the risk of cardiovascular disease. Nuts are high in zinc and magnesium. Diets deficient in zinc can lower testosterone levels. Zinc supplements have also been shown to support testosterone in zinc-deficient adults. Also, oral zinc supplements support total and free testosterone levels in elite athletes after exhaustion exercise. (Neuro Endocrin, 2006.)* Studies have shown that oleuropein, a polyphenol in olive oil, has aromatase-inhibiting properties, which might support testosterone levels. Also, the polyphenols found in red wine are natural aromatase inhibitors that decrease serum estrogen levels. Don’t overdo the red wine – it contains alcohol, which can lower testosterone and increase aromatase and estrogen levels! Red wine polyphenol supplements might be an alcohol-free alternative*. Alcohol can act as an aromatase stimulator and increase estrogen levels. Moderate consumption of red wine, one to two glasses per day, has a favorable effect on testosterone, unlike other alcoholic beverages, because of red wine’s polyphenol content. The polyphenol content of red wine is one of the health benefits of the Mediterranean Diet. A breakthrough study in the American Heart Association journal Circulation conducted at Ben-Gurion University of the Negev and Harvard University, compared body fat. Researchers compared a low-fat diet to a low-carb Mediterranean Diet. The low-carb Mediterranean Diet is more effective for reducing body fat, particularly visceral (abdominal) body fat. The diet study used magnetic resonance imaging (MRI) technology for the first time, measuring changes in body fat and organ fat during 18 months on a Mediterranean low-carb diet with moderate physical exercise. This is the best approach to date for measuring body fat compared to weighing people, as opposed to diet and exercise. The scale, skinfold calipers and underwater weighing aren’t giving you the whole picture. The Mediterranean Diet is significantly superior to a low-fat diet in decreasing fat storage including visceral (deep abdominal) liver and heart fat. High visceral fat has been shown to increase metabolic syndrome, inflammation, cardiovascular disease and diabetes. Losing deep subcutaneous visceral fat as well as hepatic (liver) fat was shown to improve lipid sensitivity and lipid profiles. The low-carb Mediterranean diet is higher in monounsaturated fats from olive oil that can raise testosterone, while low-fat diets lower testosterone production. So not only is the low-carb Mediterranean Diet ideal for weight loss, but you get healthier fats from monounsaturated fats such as olive oil and nuts, which can support testosterone. According to a most recent study in the prestigious NEJM, (New England Journal of Medicine, June 13, 2018) persons at high cardiovascular risk, the incidents of major cardiovascular events was lower among those on a Mediterranean Diet supplemented with extra-virgin-olive-oil or nuts compared to a reduced fat diet.* So, if you want to support testosterone and enhance fat loss, follow the low-carb Mediterranean Diet. * Recently, a study published in JAMA on April 25, 2018 found that the Mediterranean diet fights against frailty. It’s just another study that supports the Mediterranean diet to preserve lean body mass and health during aging. It’s also especially important to follow a high-intensity resistance training program for supporting testosterone and enhancing lean body mass. Don’t overdo long periods of cardiovascular, aerobic exercise as it can lower testosterone and encourage overtraining. Overtraining can lower testosterone and raise cortisol. Rest and recovery are important in supporting normal, healthy testosterone levels.* Coffee is also good for supporting testosterone. A Harvard University study led by Nichole Wedick showed that men who drink caffeinated coffee show increases in total testosterone and decreases in free-estradiol (estrogen). Coffee is loaded with phenolic antioxidants that act as natural aromatase inhibitors, which prevent the conversion of testosterone to estrogen (Nutrition Journal, 2012). Among men, consumption of caffeinated coffee has been shown to raise testosterone levels and decrease total estrogen and estradiol levels.* *These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease. References: Liu, TC., Lin, CH., Huang, CY. et al. Effect of acute DHEA administration on free testosterone in middle-aged and young men following high-intensity interval training. Eur J Appl Physiol (2013) 113(7): 1783-92. https://doi.org/10.1007/s00421-013-2607-x Villareal DT, Holloszy JO. Effect of DHEA on Abdominal Fat and Insulin Action in Elderly Women and Men A Randomized Controlled Trial. JAMA. 2004;292(18):2243–2248. doi:10.1001/jama.292.18.2243 Tarig Elraiyah, Mohamad Bassam Sonbol, Zhen Wang, Tagwa Khairalseed, Noor Asi, Chaitanya Undavalli, Mohammad Nabhan, Osama Altayar, Larry Prokop, Victor M. Montori, Mohammad Hassan Murad; The Benefits and Harms of Systemic Dehydroepiandrosterone (DHEA) in Postmenopausal Women With Normal Adrenal Function: A Systematic Review and Meta-analysis, The Journal of Clinical Endocrinology & Metabolism, Volume 99, Issue 10, 1 October 2014, Pages 3536–3542, https://doi.org/10.1210/jc.2014-2261 Collomp, C. Buisson, F. Lasne, R. Collomp, DHEA, physical exercise and doping, The Journal of Steroid Biochemistry and Molecular Biology, Volume 145, 2015, Pages 206-212, Luboslav Stárka, Michaela Dušková, Martin Hill, Dehydroepiandrosterone: A neuroactive steroid, The Journal of Steroid Biochemistry and Molecular Biology, Volume 145, 2015, Pages 254-260, ISSN 0960-0760, https://doi.org/10.1016/j.jsbmb.2014.03.008. Claes Ohlsson, Liesbeth Vandenput, Åsa Tivesten, DHEA and mortality: What is the nature of the association? The Journal of Steroid Biochemistry and Molecular Biology,Volume 145, 2015, Pages 248-253, ISSN 0960-0760, https://doi.org/10.1016/j.jsbmb.2014.03.006. Giovanni Corona, Giulia Rastrelli, Vito A. Giagulli, Annamaria Sila, Alessandra Sforza, Gianni Forti, Edoardo Mannucci, Mario Maggi; Dehydroepiandrosterone Supplementation in Elderly Men: A Meta-Analysis Study of Placebo-Controlled Trials, The Journal of Clinical Endocrinology & Metabolism, Volume 98, Issue 9, 1 September 2013, Pages 3615-3626, https://doi.org/10.1210/jc.2013-1358 Jankowski, C.M., Gozansky, W.S., Van Pelt, R.E., Wolfe, P., Schwartz, R.S. and Kohrt, W.M. (2011), Oral dehydroepiandrosterone replacement in older adults: effects on central adiposity, glucose metabolism and blood lipids. Clinical Endocrinology, 75: 456-463. doi:10.1111/j.1365-2265.2011.04073.x Joanna Karbowska, Zdzislaw Kochan. Fat-reducing effects of dehydroepiandrosterone involve upregulation of ATGL and HSL expression, and stimulation of lipolysis in adipose tissue, Steroids, Volume 77, Issue 13, 2012, Pages 1359-1365, ISSN 0039-128X, https://doi.org/10.1016/j.steroids.2012.08.002. A J Morales, J J Nolan, J C Nelson, S S Yen. 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